綜.合類相同項(xiàng)單選集考試卷模擬考試題

1、姓名：_ 班級(jí)：_ 學(xué)號(hào)：_-密-封 -線- 綜.合類相同項(xiàng)單選集考試卷模擬考試題考試時(shí)間：120分鐘 考試總分：100分題號(hào)一二三四五總分分?jǐn)?shù)遵守考場(chǎng)紀(jì)律，維護(hù)知識(shí)尊嚴(yán)，杜絕違紀(jì)行為，確?？荚嚱Y(jié)果公正。1、eat more, weigh less, live longerclever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.ronald kahn at harvard medical school in b

2、oston, us, and his colleagues have been able to extend the lifespan (壽命) of mice by 18 percent by blocking the rodents (嚙齒動(dòng)物) increase of fat in specific cells. this suggests that thinnessand not necessarily diet promotes long life in “calorie (熱量單位, 卡) restricted” animals.“its very cool work, “says

3、 aging researcher cynthia kenyon of the university of california, san francisco. “these mice eat all they want, lose weight and live longer. its like heaven.”calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. whether this works in humans is still un

4、known, partly because few people are willing to submit to such a strict diet.but many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. one theory is that eating less reduces the increase of harmful things that can da

5、mage cells. but kahns team wondered whether the animals simply benefit by becoming thin.to find out, they used biology tricks to disrupt the insulin (胰島素) receptor (受體) gene in lab micebut only in their fat cells. “since insulin is needed to help fat ceils store fat, these animals were protected aga

6、inst becoming fat,” explains kahn.this slight genetic change in a single tissue had dramatic effects. by three months of age, kahns modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.in addition, thei

7、r lifespan increased. the average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. after three years, all the control mice had died, but one-quarter of the modified rodents were still alive.“that they get these effects by just manipulating the fat cells is contro

8、versial,” says leonard guarente of the massachusetts institute of technology, who studies calorie restriction and aging.but guarente says kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. “it might be the same effect or there might be two

9、 routes to long life,” he points out, “and that would be very interesting./ronald kahn and his colleagues can make mice live longer by （ ）. （ ）a.offering them less foodb.giving them a balanced dietc.disrupting the specific genes in their fat cellsd.preventing them growing larger2、eat more, weigh les

10、s, live longerclever genetic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.ronald kahn at harvard medical school in boston, us, and his colleagues have been able to extend the lifespan (壽命) of mice by 18 percent by blocking the rodents (嚙齒動(dòng)

11、物) increase of fat in specific cells. this suggests that thinnessand not necessarily diet promotes long life in “calorie (熱量單位, 卡) restricted” animals.“its very cool work, “says aging researcher cynthia kenyon of the university of california, san francisco. “these mice eat all they want, lose weight

12、 and live longer. its like heaven.”calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.but many researchers hope they will be able to

13、 trigger the same effect with a drug once they understand how less food leads to a longer life. one theory is that eating less reduces the increase of harmful things that can damage cells. but kahns team wondered whether the animals simply benefit by becoming thin.to find out, they used biology tric

14、ks to disrupt the insulin (胰島素) receptor (受體) gene in lab micebut only in their fat cells. “since insulin is needed to help fat ceils store fat, these animals were protected against becoming fat,” explains kahn.this slight genetic change in a single tissue had dramatic effects. by three months of ag

15、e, kahns modified mice had up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.in addition, their lifespan increased. the average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. aft

16、er three years, all the control mice had died, but one-quarter of the modified rodents were still alive.“that they get these effects by just manipulating the fat cells is controversial,” says leonard guarente of the massachusetts institute of technology, who studies calorie restriction and aging.but

17、 guarente says kahn has yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. “it might be the same effect or there might be two routes to long life,” he points out, “and that would be very interesting./according to the passage, we do not know whether

18、 humans will benefit from taking in fewer calories partly because （ ）. （ ）a.humans, worms and rodents are differentb.most people are not willing to be put on a strict dietc.the effect is not knownd.genetic changes in tissues can not be performed on humans3、eat more, weigh less, live longerclever gen

19、etic detective work may have found out the reason why a near-starvation diet prolongs the life of many animals.ronald kahn at harvard medical school in boston, us, and his colleagues have been able to extend the lifespan (壽命) of mice by 18 percent by blocking the rodents (嚙齒動(dòng)物) increase of fat in sp

20、ecific cells. this suggests that thinnessand not necessarily diet promotes long life in “calorie (熱量單位, 卡) restricted” animals.“its very cool work, “says aging researcher cynthia kenyon of the university of california, san francisco. “these mice eat all they want, lose weight and live longer. its li

21、ke heaven.”calorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.but many researchers hope they will be able to trigger the same effect

22、 with a drug once they understand how less food leads to a longer life. one theory is that eating less reduces the increase of harmful things that can damage cells. but kahns team wondered whether the animals simply benefit by becoming thin.to find out, they used biology tricks to disrupt the insuli

23、n (胰島素) receptor (受體) gene in lab micebut only in their fat cells. “since insulin is needed to help fat ceils store fat, these animals were protected against becoming fat,” explains kahn.this slight genetic change in a single tissue had dramatic effects. by three months of age, kahns modified mice h

24、ad up to 70 percent less body fat than normal control mice, despite the fact that they ate 55 percent more food per gram of body weight.in addition, their lifespan increased. the average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. after three years, all the

25、control mice had died, but one-quarter of the modified rodents were still alive.“that they get these effects by just manipulating the fat cells is controversial,” says leonard guarente of the massachusetts institute of technology, who studies calorie restriction and aging.but guarente says kahn has

26、yet to prove that the same effect is responsible for increased lifespan in calorie-restricted animals. “it might be the same effect or there might be two routes to long life,” he points out, “and that would be very interesting./what does the last sentence in the third paragraph imply（ ）a.people like

27、 to lose weight, but they do not like to eat less.b.people want to go to heaven,but they do not want to die.c.mice will go to heaven if they lose weight.d.mice enjoy losing weight.4、eat more, weigh less, live longerclever genetic detective work may have found out the reason why a near-starvation die

28、t prolongs the life of many animals.ronald kahn at harvard medical school in boston, us, and his colleagues have been able to extend the lifespan (壽命) of mice by 18 percent by blocking the rodents (嚙齒動(dòng)物) increase of fat in specific cells. this suggests that thinnessand not necessarily diet promotes

29、long life in “calorie (熱量單位, 卡) restricted” animals.“its very cool work, “says aging researcher cynthia kenyon of the university of california, san francisco. “these mice eat all they want, lose weight and live longer. its like heaven.”calorie restriction dramatically extends the lifespan of organis

30、ms as different as worms and rodents. whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.but many researchers hope they will be able to trigger the same effect with a drug once they understand how less food leads to a longer life. one

31、 theory is that eating less reduces the increase of harmful things that can damage cells. but kahns team wondered whether the animals simply benefit by becoming thin.to find out, they used biology tricks to disrupt the insulin (胰島素) receptor (受體) gene in lab micebut only in their fat cells. “since i

32、nsulin is needed to help fat ceils store fat, these animals were protected against becoming fat,” explains kahn.this slight genetic change in a single tissue had dramatic effects. by three months of age, kahns modified mice had up to 70 percent less body fat than normal control mice, despite the fac

33、t that they ate 55 percent more food per gram of body weight.in addition, their lifespan increased. the average control mouse lived 753 days, while the thin rodents averaged a lifespan of 887 days. after three years, all the control mice had died, but one-quarter of the modified rodents were still a

34、live.“that they get these effects by just manipulating the fat cells is controversial,” says leonard guarente of the massachusetts institute of technology, who studies calorie restriction and aging.but guarente says kahn has yet to prove that the same effect is responsible for increased lifespan in

35、calorie-restricted animals. “it might be the same effect or there might be two routes to long life,” he points out, “and that would be very interesting./the average modified mouse lived （ ）. （ ）a.3 yearsb.753 daysc.more than 3 yearsd.887 days5、eat more, weigh less, live longerclever genetic detectiv

36、e work may have found out the reason why a near-starvation diet prolongs the life of many animals.ronald kahn at harvard medical school in boston, us, and his colleagues have been able to extend the lifespan (壽命) of mice by 18 percent by blocking the rodents (嚙齒動(dòng)物) increase of fat in specific cells.

37、 this suggests that thinnessand not necessarily diet promotes long life in “calorie (熱量單位, 卡) restricted” animals.“its very cool work, “says aging researcher cynthia kenyon of the university of california, san francisco. “these mice eat all they want, lose weight and live longer. its like heaven.”ca

38、lorie restriction dramatically extends the lifespan of organisms as different as worms and rodents. whether this works in humans is still unknown, partly because few people are willing to submit to such a strict diet.but many researchers hope they will be able to trigger the same effect with a drug

39、once they understand how less food leads to a longer life. one theory is that eating less reduces the increase of harmful things that can damage cells. but kahns team wondered whether the animals simply benefit by becoming thin.to find out, they used biology tricks to disrupt the insulin (胰島素) receptor (受體) gene in lab m